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Trombosis, Emboli, Infark
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Trombosis
• Adlh pembentukan b lood c lo t (t rom bus )
dlm PD/jantung• Peristiwa fisiologis pd individu hidup• b lood c lo t (t rom bus ) → mhalangi aliran
darah
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• Trombosis → awal dr mekanisme b l o o dc lo t t ing thd respon adanya kelainan dalamdarah/PD
• Mekanisme clotting → konjugasi antara platelets(trombosit) & plasma darah in conjunction withchemicals carried in blood plasma
• Konjugasi → aktifkan protrombin → trombin →katalis pbentukan fibrinogen → fibrin
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Representation of the Common Pathway
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• Ada 2 jalur :
- intrinsik- ekstrinsik
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• Faktor pembentuk fibrin → prokoagulan
• Untuk mencegah Prokoagulan bekerjaterus menerus → tdp Ant ikoagu lan
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Penyebab Trombosis
• Hipertensi & aterosklerosis → intrinsik →krn adanya pembentukan kolagen
• Aliran darah yg tiba2 jd lambat →platelate (trombosit) kontak dg lapisanendotel → terbtk koagulasi
• Contoh → varises
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Early events in thrombosis
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Types of Thrombi
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Tipe Emboli
• Thromboembolism (embolism of thrombus or blood clot)
• Fat embolism (embolism of fat droplets)
• Air embolism (embolism of air bubbles)
• Gas embolism (embolism of blood gas such as oxygen, nitrogen, helium, etc)
• Septic embolism (embolism of pus containing bacteria)
• Tissue embolism (embolism of small fragments of tissue)
• Foreign body embolism (embolism of foreign materials such as talc and other
tiny objects)
• Amniotic fluid embolism (amniotic fluid, fetal cells, hair or other debris entersthe mother's blood stream via the placental bed of the uterus and triggers anallergic reaction)
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Tipe Jalur Emboli
• Anterograde
• Retrograde
• Paradoxical
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Infark
• Lanjutan dari trombus & emboli → bilaembolus mhalangi PD ke organ/jaringan→ infark
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Normal Infark
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